I moved to Krill oil for personal reasons. It is a highly solubilized type of EPA and DHA... so less is needed because of its better absorption. It also has an antioxidant naturally in it (astaxanthin).
I use 2 MegaReds a day.
http://www.consumerreports.org/cro/2...-oil/index.htm
And this is a new paper:
Quote:
Eur J Nutr. 2013 Jun;52(4):1315-25. doi: 10.1007/s00394-012-0441-2. Epub 2012 Aug 25.
Krill oil versus fish oil in modulation of inflammation and lipid metabolism in mice transgenic for TNF-α.
Vigerust NF, Bjørndal B, Bohov P, Brattelid T, Svardal A, Berge RK.
Source
Institute of Medicine, University of Bergen, Bergen, Norway. Natalya.Vigerust@med.uib.no
Abstract
PURPOSE:
Biological effects of marine oils, fish oil (FO) and krill oil (KO), are mostly attributed to the high content of n-3 polyunsaturated fatty acids (n-3 PUFAs), predominantly eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA). The study was aimed to investigate the influence of FO and KO on lipid homeostasis and inflammation in an animal model of persistent low-grade exposure to human tumor necrosis factor α (hTNF-α) and to evaluate whether these effects depend on the structural forms of EPA and DHA [triacylglycerols (TAG) vs. phospholipids].
METHODS:
Male C57BL/6 hTNF-α mice were fed for 6 weeks a high-fat control diet (24.50 % total fats, w/w) or high-fat diets containing either FO or KO at similar doses of n-3 PUFAs (EPA: 5.23 vs. 5.39 wt%, DHA: 2.82 vs. 2.36 wt% of total fatty acids).
RESULTS:
We found that KO, containing bioactive n-3 PUFAs in the form of phospholipids, was capable of modulating lipid metabolism by lowering plasma levels of TAG and cholesterol and stimulating the mitochondrial and peroxisomal fatty acid β-oxidation, as well as improving the overall carnitine turnover. Though the administration of FO was not as effective as KO in the lowering of plasma TAG, FO significantly improved the levels of all cholesterol classes in plasma. Except from the increase in the levels of IL-17 in FO-fed mice and a trend to decrease in MCP-1 levels in KO-fed animals, the levels of pro-inflammatory cytokines were not substantially different between treatment groups.
CONCLUSION:
Our findings demonstrate that FO and KO are comparable dietary sources of n-3 PUFAs. However, when quantitatively similar doses of n-3 PUFAs are administered, KO seems to have a greater potential to promote lipid catabolism. The effect of dietary oils on the levels of inflammatory markers in hTNF-α transgenic mice fed a high-fat diet needs further investigations.
PMID:
22923017
[PubMed - indexed for MEDLINE]
|
from
http://www.ncbi.nlm.nih.gov/pubmed/22923017
and this is interesting too:
http://www.ncbi.nlm.nih.gov/pubmed/23876229
Phospholipids is what Krill oil has.
Fish oil or Krill oil...it is a personal choice. But both help with inflammation.
__________________
All truths are easy to understand once they are discovered; the point is to discover them.-- Galileo Galilei
************************************
.
Weezie looking at petunias 8.25.2017
****************************
These forums are for mutual support and information sharing only. The forums are not a substitute for medical advice, diagnosis or treatment provided by a qualified health care provider. Always consult your doctor before trying anything you read here.