Dearest Neurotalk friends,

I was doing 'more research' on Copper Deficiency recently and found two interesting (to me!) articles:

One was at a Mayo Clinic site, and connected copper deficiency to recovery from accute inflammation episodes, just like the ones I had constantly as the reaction to my 10 Hizentra infusions and one Gammagard infusion:

Because ceruloplasmin is an acute phase reactant, urine copper is elevated during acute inflammation. During the recovery phase, urine copper is usually below normal, reflecting the expected physiologic response to replace the copper that was depleted during inflammation.

http://www.mayomedicallaboratories.c...erpretive/8590

The second was a New York Times article on urine tsting for Copper. What made that aritcle interesting is that is doesn't even mention copper deficiency. The only way is describes abnormal test results is as an excess of copper in your urine.

Copper deficiency is so rare, it doesn't rate a mention in this article.

But I really think I may be on to something with the relatonship between copper deficiency as a result of accute inflammation episodes.

I did send an email to my Neurologist about this, too.

Hugs, Elaine