This is the final paragraph before the listed references (this artical was published in jan 2000)

CS, IS and MS may be distinct phenomena and it is
possible to hypothesize different underlying causes for
each. However, it is also possible that all three kinds of
spread may be expressions of a generalized disorder of
CNS regulation of neurogenic infammation. Neurogenic
infammation is almost certainly a multi-dimensional
phenomenon involving, for example, the generation of antidromic
impulses in primary afferent sensory neurons (resulting
in the peripheral release of neuropeptides like substance
P and calcitonin gene-related peptide), an interaction
between primary afferent terminals and the terminals of
postganglionic sympathetic efferents, and CNS regulation
of the immune system's contribution to infammation
(Eliav et al., 1999). Aberrant CNS regulation of neurogenic
inflammation may account for the initial appearance of
CRPS-I/RSD, for all three kinds of spread, for the movement
disorder (Schwartzman and Kerrigan, 1990; Bhatia et
al., 1993; Veldman et al., 1993; Baron et al., 1996), and for
the dysregulation of vasomotor and sudomotor function
(Kozin et al., 1976a,b; JaČnig, 1985; Schwartzman and Kerrigan,
1990; Bej and Schwartzman, 1991; Herrick et al., 1994;
Baron and Maier, 1996; Kurvers et al., 1996; Birklein et al.,
1998; Wasner et al., 1999).: