Thread: Hearing loss
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Old 08-18-2014, 09:13 AM
lisabeth36 lisabeth36 is offline
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Join Date: Aug 2014
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8 yr Member
lisabeth36 lisabeth36 is offline
New Member
 
Join Date: Aug 2014
Posts: 1
8 yr Member
Default Hearing Loss and Myesthenia Gravis

There are several articles when you google on this issue. I woke up this morning with 100% hearing loss in my left ear. I can't hear even tapping on my outer ear or head on that side, nothing, nada. I have a Doc. appt. this afternoon. I am in a mild state of panic. :-) Here is one article I read this morning in pubmed.

Abstract
OBJECTIVE:
Acetyl choline (ACh) is the main neurotransmitter of the efferent auditory system. This study is aimed to evaluate cochlear function in myasthenia gravis (MG), a neuromuscular transmission disorder caused by ACh receptor autoantibodies.
METHODS:
This prospective study included 16 myasthenic patients, tested audiologically twice, first after improvement from myasthenic crisis or acute oropharyngeal dysfunction (1 week from admission) and then 2 months later. We detected the effect of contralateral acoustic stimulation (CAS) on patients' transient and distortion product otoacoustic emissions (TEOAE and DPOAE).
RESULTS:
Compared with controls, patients reported significant reduction in overall echo response and amplitude of TEOAEs at 1-2 kHz and at 1-6 kHz of DPOAE with marked reduction at 5 kHz. In the control group, CAS produced amplitude reduction in TEOAEs and DPOAEs at 1-4 kHz. Utilizing masking effect, patients reported amplitude reduction in TEOAEs at 1.5-4 kHz while DPOAEs did not reach significant level except at 1.5 and 5 kHz. After 2 months, no changes were observed compared with early assessment.
CONCLUSIONS:
It is clear that disease progression is associated with irreversible cochlear damage. Lack of improvement in patients' emissions despite partial non-audiometric improvement in relation to receptors needs to be considered.
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