Quote:
Originally Posted by lurkingforacure
I would think another option would be to find out why/what causes the alphasynuclein to "pathologically morph" in the first place. How is it different from the alphasyn in people without PD? At what point does it begin to morph? There are a lot of questions that could be asked that might get to the actual heart of the matter, in addition to trying to stop it from spreading once it has already morphed.
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There are several reasons. Mutations in alpha syn and over expression of the gene. Chemical insults to the protein, inflammation and aggregation of protein s trands are a few that cause alpha-syn to morph.
Curcumin is supposed stop this morphing atleast in vitro.