As a follow-up to a previous post in this thread, I have found some interesting
Medline studies that indicate/confirm increased toxic metal absorption/toxicity when nutritional minerals are deficient/depleted. As mentioned before, this would appear to be a very good explanation for the previous hair test results. Interesting quotes from the referenced abstracts are displayed below. By the way, I have now increased the dosage of the
EDTA chelator to 1 teaspoon/day (still a rather low dose) per my doctor’s instructions:
http://www.ncbi.nlm.nih.gov/sites/en...um&query_hl=19 (The full text version of this study also references certain vitamins which, when depleted, also lead to increased toxic metal absorption/toxicity.):
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Based on data from mechanistic studies, the ability of micronutrients to modulate the toxicity of metals is indisputable.
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Therefore, people eating a diet deficient in micronutrients will be predisposed to toxicity from nonessential metals.
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http://www.ncbi.nlm.nih.gov/sites/en...sum&query_hl=3
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Cadmium, lead, mercury, and aluminum are toxic metals that may interact metabolically with nutritionally essential metals.
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http://www.ncbi.nlm.nih.gov/sites/en...sum&query_hl=3
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However, the toxic effects of these metals may be mediated or enhanced by interactions or deficiencies of nutritionally essential metals.
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http://www.ncbi.nlm.nih.gov/sites/en...sum&query_hl=1
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In general, a deficiency of these essential elements increases toxicity of heavy metals, whereas an excess appears to be protective. While most of the observations are on laboratory animals, limited human data are in agreement with the results of animal experiments. These suggest that the dietary presence of the essential elements may contribute to the protection of man and animal from the effects of heavy metal exposure, while their deficiency may increase toxicity.
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http://www.ncbi.nlm.nih.gov/sites/en...sum&query_hl=2 (This is an animal study, but probably applicable to humans as well.):
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Since the toxicity of one metal or metalloid can be dramatically modulated by the interaction with other toxic or essential metals, studies addressing the chemical interactions between trace elements are increasingly important.
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These interactions probably indicate that mineral balance in the body is regulated by important homeostatic mechanisms in which toxic elements compete with the essential metals, even at low levels of metal exposure.
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http://www.ncbi.nlm.nih.gov/sites/en...sum&query_hl=1
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Oral ingestion of these toxic metals perturbs the metabolism of essential elements, especially zinc (Zn), copper (Cu), iron (Fe), and selenium (Se).
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http://www.ncbi.nlm.nih.gov/sites/en...sum&query_hl=1
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Although these metals are known to produce their toxic effects on a variety of body systems, much emphasis has been placed on their effects on the nervous system owing to apparent association of relatively low or "subclinical" levels of metallic exposure with behavioral and psychological disorders.
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The net toxic manifestations produced by multiple exposure should, therefore, be different from those produced by a single factor as the result of their additive, synergistic or antagonistic action. Even though a metal may not exist in sufficient amounts to cause any disability, the toxicity could result when a second factor is also present.
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