Quote:
Originally Posted by mrsD
Everyone is different. B6, has to be activated in the body as Pyroidoxine itself is not biologically active in the tissues. This conversion is dependent on B2 (riboflavin) and other biological factors, which can vary from person to person.
Deficiency as well as long term toxicity of high dose supplements both give clinical neurological signs so diagnosis is difficult. Even the testing may be faulty, so that furthers poor diagnostic accuracy.
Here is a good monograph about B6:
Dietary Supplement Fact Sheet: Vitamin B6 — Health Professional Fact Sheet
I think others here could benefit from some data, links that show the dosing in studies that becomes problematic. Anecdotal comments from patients, is not that helpful in the long run, IMO. |
also, the long term supplementation that you talk about is also incorrect. There are some who only took it for a week or two and now have neuropathy all over their body. Everything you are referencing is from studies that date multiple decades ago. Take a look at the b6 boards and all the people suffering from its toxicity and you will see that none of what you are saying is correct.