The only thing that I can find is this very technical paper.
What they found is that lenalidomide (a thalidomide analog) blocked progression of EAE, which is a mouse model of MS.
They suggest that lenalidomide (and maybe other thalidomide analogs?) is a promising therapeutic drug candidate for attenuating inflammatory responses and reducing neuronal demyelination in MS.
Lenalidomide regulates CNS autoimmunity by promoting M2 macrophages polarization | Cell Death & Disease