Cortisol counteracts insulin, contributes to hyperglycemia-causing hepatic gluconeogenesis and inhibits the peripheral use of glucose (insulin resistance) by decreasing the translocation of glucose transporters (especially GLUT4) to the cell membrane.
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Originally Posted by alice md
Insulin secretion as a response to glucose is a complex biological process.
Theoretically, increased levels of acetyl-choline could increase this response, in certain conditions.
This means that in most people it would not have this effect. But, it could become significant if there is also some underlying sub-clinical metabolic abnormality.
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