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Originally Posted by KimS
II do, however, question the claim that Haas or Gottschall ever said that celiac could be cured... but I have read that it is the original celiac diet... that grains were not supposed to ever be introduced back into the diet. I don't remember reading that celiacs could go back to eating a 'normal' diet.
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I think this would pertain to the early 1950s - it wasn't until 1951 that the Drs. Haas published their diet as a treatment and cure for celiac and cystic fibrosis of the pancreas. My interpretation of what is said here is that people who had been diagnosed with celilac were considered healed as a result of the inherent lectin restrictions of the then new SCDiet, which brings into question whether their original diagnoses were correct. Did they have celiac, or gluten intolerance, or were they in fact suffering from
lectin intolerance? Again, this is what started me thinking that perhaps a certain percentage of diagnosed
gluten intolerators (that is, not possesing either DQ2 or DQ8) are really
lectin intolerators. If this is the case, the very stringently defined diagnosis of villous atrophy and the possession of celiac genes is actually accurate.
Of course, this now brings into question our concept of what we experience as exclusively being a gluten intolerance. Do all of the genes identified as linked to gluten intolerance (not DQ2 & 8) really mean these are actually markers for
lectin intolerance? I've done a lot of searching and have yet to find that connection, possibly because it hasn't been made. Does anyone know if Fine has ever written about lectins? Could it be we're all missing a very important part of the puzzle?
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In many cases cited in the book, elimination of certain carbohydrates 'cured' diagnosed Celiacs after one year and they were able to return to eating gluten containing foods. In hindsight many of the foods eliminated in this plan are high lectin foods known to be associated with gut and systemic inflammatory reactions. Celiac-Sprue is a genetic disorder treated by elimination of offending foods. The response of some to the specific carbohydrate elimination diet would mean that the patients who responded did not have classical gluten intolerance, Celiac-Sprue, which requires life long elimination of gluten/gliadin. It suggests that other lectins may cause similar symptoms and overlapping diagnostic and treatment difficulties.
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