You'll have to decide for yourself.
I am not in reality advocating other statins over Lipitor. Only pointing out that just because they are now generic, they are IGNORED by doctors for the most part unless insurances force their use. In fact Pfizer has cleverly insinuated a study to convince even the GODS of insurance payout that Lipitor is BETTER than other statins. Yes they have. The only statin that crosses into the brain well is Zocor. (this may be good or bad depending on what happens to the patient in the final analysis. But it is a provable fact).
So rather than research the drugs, you should look into why your cholesterol is high to begin with. 1) goes up for almost every human as they age you know. 2) is there some feedback loop connected to your pituitary problem? Cholesterol is a precursor for making all hormones you know. So does cholesterol go up, because we are no longer making estrogen etc?
The studies are just not good enough...
http://www.cholesterol-and-health.co...es-Stroke.html
See how you feel after reading this article! then read this new article on the toxicity of statins to mitochondria:
Quote:
Mitochondrial impairment by PPAR agonists and statins
Toxicol Appl Pharmacol. 2007 Jun 21; : 17658574 PubMed
Mitochondrial impairment by PPAR agonists and statins identified via immunocaptured OXPHOS complex activities and respiration.
MitoSciences, Inc., 1850 Millrace Drive, Eugene, OR 97403, USA
Sashi Nadanaciva , James A Dykens , Autumn Bernal , Roderick A Capaldi , Yvonne Will
Mitochondrial impairment is increasingly implicated in the etiology of toxicity caused by some thiazolidinediones, fibrates, and statins. We examined the effects of members of these drug classes on respiration of isolated rat liver mitochondria using a phosphorescent oxygen sensitive probe and on the activity of individual oxidative phosphorylation (OXPHOS) complexes using a recently developed immunocapture technique. Of the six thiazolidinediones examined, ciglitazone, troglitazone, and darglitazone potently disrupted mitochondrial respiration. In accord with these data, ciglitazone and troglitazone were also potent inhibitors of Complexes II+III, IV, and V, while darglitazone predominantly inhibited Complex IV. Of the six statins evaluated, lovastatin, simvastatin, and cerivastatin impaired mitochondrial respiration the most, with simvastatin and lovastatin impairing multiple OXPHOS Complexes. Within the class of fibrates, gemfibrozil more potently impaired respiration than fenofibrate, clofibrate, or ciprofibrate. Gemfibrozil only modestly inhibited Complex I, fenofibrate inhibited Complexes I, II+III, and V, and clofibrate inhibited Complex V. Our findings with the two complementary methods indicate that (1) some members of each class impair mitochondrial respiration, whereas others have little or no effect, and (2) the rank order of mitochondrial impairment accords with clinical adverse events observed with these drugs. Since the statins are frequently co-prescribed with the fibrates or thiazolidinediones, various combinations of these three drug classes were also analyzed for their mitochondrial effects. In several cases, the combination additively uncoupled or inhibited respiration, suggesting that some combinations are more likely to yield clinically relevant drug-induced mitochondrial side effects than others.
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As long as the studies funded by the drug companies themselves are taken as truth, the waters will always be muddy, IMO.
removing personal information
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All truths are easy to understand once they are discovered; the point is to discover them.-- Galileo Galilei
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Weezie looking at petunias 8.25.2017
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