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Old 10-01-2006, 02:12 PM
jccgf jccgf is offline
Senior Member (jccglutenfree)
 
Join Date: Aug 2006
Location: Wisconsin
Posts: 1,581
15 yr Member
jccgf jccgf is offline
Senior Member (jccglutenfree)
 
Join Date: Aug 2006
Location: Wisconsin
Posts: 1,581
15 yr Member
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Hi Diana,

Have you read this site yet, about the relationship between gluten and GERD?
http://www.cryingoverspiltmilk.co.nz...uxdisease!.htm

Anne just posted some info the other day about Dr. Rodney Ford here~ http://forums.braintalk2.org/showthread.php?t=2065


The positive IgG testing suggests some degree of wheat sensitivity. There is a food allergy page in The Gluten File linked below, that has some info on IgG/IgE testing and what it means. Classic allergy is based on IgE reactions. IgG reactions are considered 'delayed' food allergy. Some of the allergy sites listed there give good explanations. Generally, mainstream allergy testing only looks for IgE allergy, but it appears that the idea of IgG allergy is catching on.

There is also a page about gluten sensitivity vs. celiac disease with links to websites of doctors who are more open to the idea that gluten sensitivity can cause problems in people who don't have celiac disease. The IgG antibodies seem to be the only indicator some of us have.

Here are a few things about Gerd and asthma. They can also be related.

Quote:

Gastroesophageal reflux (GER) is a potential trigger of asthma. The esophagus and lung interact through a variety of mechanisms. Esophageal acid-induced bronchoconstriction can be provoked by a vagally mediated reflex, whereby acid in the distal esophagus produces airway responses; by neural enhancement of bronchial reactivity, whereby esophageal acid augments airway hyperresponsiveness; or by microaspiration, in which small amounts of esophageal acid in the upper airway cause significant airway responses. Interestingly, even in the microaspiration model, the vagus nerve plays a significant role. Neurogenic inflammation in the lung may occur with either vagally mediated mechanisms or with microaspiration. The prevalence of reflux symptoms, esophagitis, and abnormal esophageal acid contact time is higher in patients with asthma than in control populations. Potential mechanisms, whereby asthma may predispose to the development of GER, include autonomic dysregulation, an increased pressure gradient differential between the thorax and the abdomen, a high prevalence of hiatal hernia, alterations in crural diaphragm function, and bronchodilator medication use. Further research will help define how the esophagus and lung interact.
Gastroesophageal reflux, asthma, and mechanisms of interaction.
PMID: 11749917

Gastroesophageal reflux (GER) is a factor often neglected in the etiopathogenesis of asthma. The estimated incidence of GER in asthmatic children reaches 50-60% and is higher than in the general population. GER may accompany typical symptoms: hoarseness, sore throat, thoracic pain, cough or wheezing. GER may not only aggravate the course of bronchial obstruction, but may also cause it, or trigger obstruction due to other factors. Asthma and GER coincidence has been acknowledged for many years.
Asthma and gastroesophageal reflux in children. PMID: 11887043 (free full text available as download)
Last but not least, have you ever read about or had your daughter tested for Pyroluria? FJ on the TS board has posted about this (and is where I heard about it), so you may know all about it. There is also a page about it in The Gluten File, linked down near the bottom of the right bar. It has some increased association in certain populations, including autism, OCD, and is associated with gluten/casein sensitivities.

I may have more later, and I will have to reread your post more carefully. I am in a hurry, but this is what I could find quickly. Sorry to just dump and run. Oh..you might also check out TGF page called Not Celiac? It reviews some other information on food allergy, protein intolerance, etc.

Cara
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