If the hypothesis is correct, then we should look for two things - those that block the immune response of inflammation and those that block the stress response.

1: Pharmazie. 2007 Dec;62(12):937-42.
Neuroprotective effect of curcumin is mainly mediated by blockade of microglial
cell activation.
Lee HS, Jung KK, Cho JY, Rhee MH, Hong S, Kwon M, Kim SH, Kang SY.
"Indeed, curcumin blocked the production of pro-inflammatory and cytotoxic mediators such as NO, TNF-alpha, IL-1alpha, and IL-6 produced from Abeta(25-35)/IFN-gamma- and LPS-stimulated microglia, in a dose-dependent manner. Therefore, our results suggest that curcumin-mediated neuroprotective effects may be mostly due to its anti-inflammatory effects."
http://www.ncbi.nlm.nih.gov/pubmed/1...ubmed_RVDocSum

1: Curr Pharm Des. 2007;13(18):1925-8.
Inflammation in Parkinson's diseases and other neurodegenerative diseases: cause
and therapeutic implications.
Wilms H, Zecca L, Rosenstiel P, Sievers J, Deuschl G, Lucius R.
http://www.ncbi.nlm.nih.gov/pubmed/1...ubmed_RVDocSum

1: J Neurosci Res. 2004 Dec 1;78(5):723-31.
(-)-Epigallocatechin gallate inhibits lipopolysaccharide-induced microglial
activation and protects against inflammation-mediated dopaminergic neuronal
injury. <Green Tea>
Li R, Huang YG, Fang D, Le WD.
http://www.ncbi.nlm.nih.gov/pubmed/1...ubmed_RVDocSum

1: Int Immunopharmacol. 2007 Mar;7(3):313-20. Epub 2006 Dec 1.
Differential effects of ginsenosides on NO and TNF-alpha production by
LPS-activated N9 microglia. <Ginseng>
Wu CF, Bi XL, Yang JY, Zhan JY, Dong YX, Wang JH, Wang JM, Zhang R, Li X.
http://www.ncbi.nlm.nih.gov/pubmed/1...ubmed_RVDocSum