It is your interpretation that my statement was emphatic. It is not my opinion if it is based on facts.

I stated a fact that I supported with one website and one reference from a book I own. Philpott is an MD, the other other of that book, Kalita, is a phd.

Asking me a personal question is making this personal about me. I won't dignify that question.


http://www.ceri.com/fftrypto.htm
"In addition, tryptophan catabolism in the brain and peripheral tissues produces toxic chemicals which stimulate excitatory neurotransmitter pathways."
"Given the known adverse influences of some IDO-induced tryptophan metabolites, supplementing tryptophan may be an inappropriate strategy."

Another reference to tryptophan metabolites (that are potentially toxic) but in reference to AD.

http://www.ncbi.nlm.nih.gov/entrez/q...&dopt=Abstract

One subcellular mechanism which may underlie degeneration of neurons in AD is decreased axonal transport with accumulation of enzymes and their potentially toxic metabolites in the cell body.

These three metabolites of tryptophan are toxic: hydroxykynurenine, xanthurenic acid and hydroxyanthranilic acid. Therefore tryptophan can be toxic if it is not converted properly. That toxicity depends on the pathway it takes in the brain. To avoid the possibility of toxicity there has to be enough b6 and niacin.

http://www.pubmedcentral.nih.gov/pic...6&blobtype=pdf