Quote:
Originally Posted by Dubious
Oh, I forgot to ask, respectfully of course; while I didn't look at all of your abstracts as closely as I had the time for, the ones I did look at focused only on CRPS I. An interesting hyposthesis, of course! How does CRPS II, where there is direct nerve injury that precipitated the onset of the condition, relate to the hypothesis of a causative viral agent, that in an individual with an uncompromised immune system, process this? Remember, !
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Oh, that's easy.
100% of the CRPS-2 patients were positive for parvovirus Ig(G). That said, Ig(G) titers of parvovirus Ig(G) between CRPS and controls showed no significant difference (13.3 ± 18.6 vs. 14.6 ± 35.1), while CRPS-2 patients (11/11) had a significant higher prevalence than CRPS-1 patients (17/28, 60.7%,
p < 0.05), but both groups had a higher prevalence than the controls (
p < 0.01). [G. Oliver et al. /
European Journal of Pain 11 (2007) 237–240 at 238.]
Mike