as per:
This overload stimulated production of dangerous reactive oxygen species that interfered with the ability of the mitochondria to transport sugar needed for energy production. This phenomenon could be reversed by providing substrates for energy production..."

http://todaysseniorsnetwork.com/calc...uced_death.htm

Further in the article is this notation:
"These data strongly suggest that the respiratory complexes in PINK1 deficiency are still intact and that their functional inhibition is in fact secondary to reduced substrate supply," offers Dr. Abramov

thus, the mitochondria are not diseased and the respiratory electron transport chain complexes are also functional--the substrates that constitute the complexes are decreased; ie NADH+CoQ10 for complex 1. Rotenone pesticide is the best known inhibitor of complex 1.

Link for the abstract:
http://www.cell.com/molecular-cell/a...765(09)00129-4