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Old 08-06-2009, 08:36 PM
jccgf jccgf is offline
Senior Member (jccglutenfree)
 
Join Date: Aug 2006
Location: Wisconsin
Posts: 1,581
15 yr Member
jccgf jccgf is offline
Senior Member (jccglutenfree)
 
Join Date: Aug 2006
Location: Wisconsin
Posts: 1,581
15 yr Member
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And remember... getting the level up can happen quickly... but healing takes place over months to years if nerves were damaged and need to repair.

Found another reference to nitrous oxide while checking something out for someone else:

http://emedicine.medscape.com/article/1171558-overview

Quote:
Cyanocobalamin (vitamin B 12 ) deficiency
  • About 80% of all cases are due to pernicious anemia, and another 10% are due to achlorhydria. Exposure to nitrous oxide can suddenly precipitate the deficiency, which should be considered in any patient who develops postoperative paresthesias.
  • The disease predominantly affects the spinal cord; therefore, separating the painful sensory and sensorimotor paresthesias of the peripheral neuropathy from the symptoms of spinal cord involvement is difficult.
  • Presentations vary greatly among patients.
  • The symmetric glove-and-stocking paresthesias, or tingling in the distal aspect of the toes, numbness, coldness, a pins-and-needles feeling, and occasional feelings of swelling or constriction, are slowly progressive and insidious. Symptoms progressing up the legs, occasionally affect the fingers, and culminate in weakness and spasticity.
  • In late stages, signs include moderate muscular wasting, optic atrophy, sphincter dysfunction, and mental disturbances. Examples of these disturbances are mild dementia (which is often the first symptom and clinically indistinguishable from other dementias), disorientation, depression, psychosis, and persecutory delusions.
  • The hematologic manifestation of anemia, if present, can cause weakness, light-headedness, vertigo, tinnitus, palpitations, angina, heart failure, cardiomegaly, pallor, tachycardia, and hepatosplenomegaly.
  • GI symptoms include a sore, beefy red tongue and anorexia.
  • If left untreated, the gait becomes ataxic, followed by paraplegia with spasticity and contractures.
  • The subacute combined degeneration that develops results in a severe myelopathy, involving posterior columns and lateral corticospinal tracts, with other manifestations including optic (retrobulbar) neuropathy, sensorimotor polyneuropathy, and dementia.
~

hehehe... the persecutary delusions come from doctors not knowing how to identify and treat B12 deficiency...
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