I have a theory on this, It seems that a rapid reactivation of any residual levadopa causes dyskenesia/dystonia. This rapid reactivation can be promoted by starting to eat or just in anticipation of food. Glycogen will readily bind with levodopa so we are looking for a release mechanism to break the bond between glycogen and levodopa. The theoretical solution suggests that it is related to the pancreas as digestive enzymes and other hormones (including insulin) are produced when you eat anything (this would explain the lack of consistancy about what food groups can cause this effect). The link is that most cells of the body have insulin receptors which bind the insulin to the cell and activates other receptors on the cell which are designed to absorb glucose (sugar) from the blood stream into the inside of the cell. This reduction in glucose in the blood frees the levadopa and the resultant rush causes the dyskenesia/dystonia. Theoretically the way to control it is to take a glycogen supplement to re-bind the levadopa or reduce the amount of insulin…

Of course it could be none of the above, any comments…. Im new at this.