Annie, can I ask you another question, since you've been so generous? This is another I-thought-I-understood-but-now-I'm-not-sure question.

My understanding was that we myasthenics produce normal amounts of acetylcholine, but the receptor cells that bind to it are damaged by our immune systems. So although we have normal amounts of acetylcholine, not enough of it is able to get through to the muscles. I was assuming that when the Mestinon allows the acetylcholine to hang around longer, that means the receptor cells we have that are working can do double duty, to make up for the ones that aren't working.

But, I realized I was just sort of vaguely guessing. Will you correct and fine-tune that for me, too? Is it that myasthenics produce what would be enough acetylcholine for a normal person, but not for a person with damaged muscle receptor cells?

Thanks again,

Abby