hi
thanks for reply,s

it is just I find the division of the terms unfair to the sufferer as you rightly describe shez MTBI is a better term please see cut and paste . I just feel the term syndrome seems to suggest. to the uniformed more of a psychological malady than the real injury that it is

I have been diagnosed the following is an abstract of my report


The following cognitive abilities were assessed:
• Pre-morbid intellectual and memory potential
• Neuropsychological status (dementia screening)
• Working memory ability
• Speed of information processing
The results are as follows (test description in Appendix 1):
Estimated Intellectual memory Potential
####### pre-morbid intellectual memory abilities were estimated to be within the 40 to 75 range

Neuropsychological Status
His performance on measures of attention, immediate and delayed memory, language and
visuospatial/constructional ability are summarised in a Total score that indicates overall
neurocognitive status below the 20th percentile for his age-group, and is strongly indicative
of an acquired impairment.

His most prominent deficit areas are the attentionai and memory indexes. His immediate
memory index is below normal, probably due to the result of impaired attentional/working
memory ability and on account of this poor initial processing of information, his delayed
recall index is also below average.

His language (confrontational naming and semantic fluency) and visuospatial/constructional
abilities are relatively preserved. ( my note relatively preserved dosan,t feel like it )
Working Memory
His working memory (auditory and visual) is consistent with Borderline functioning (4th
percentile) and in stark contrast with the estimated pre-morbid ability.
Speed of Information Processing
His information speed registered in the Defective range (4th percentile) and also represents
a significant change from pre-morbid functioning.
Summary
is a 47 year old man with an acquired brain injury subsequent to an assault
during December 2007, with ongoing deficits with attention, memory and an executive
function in the context of a relatively stable mood.
Recent initial neuropsychojnetry confirmed a clearly unfavourable neuropsychological profile
with deficits in attention, working memory, memory and speed'onnformation processing "-
all of which are inconsistent with pre-morbid levels of functioning.

thanks mark for you in depth answer, based on the above what class am I in MTBI or TBI 2 years plus injury ?

please note CSF leak post injury so I lost the cushion around the brain on impact

and night epilepsy





NET DEFINITION

Traumatic brain injury (TBI) can significantly affect many cognitive, physical, and psychological skills. Physical deficit can include ambulation, balance, coordination, fine motor skills, strength, and endurance. Cognitive deficits of language and communication, information processing, memory, and perceptual skills are common. Psychological status is also often altered. Adjustment to disability issues are frequently encountered by people with TBI.

Brain injury can occur in many ways. Traumatic brain injuries typically result from accidents in which the head strikes an object. This is the most common type of traumatic brain injury. However, other brain injuries, such as those caused by insufficient oxygen, poisoning, or infection, can cause similar deficits.

Mild Traumatic Brain Injury (MTBI) is characterized by one or more of the following symptoms: a brief loss of consciousness, loss of memory immediately before or after the injury, any alteration in mental state at the time of the accident, or focal neurological deficits. In many MTBI cases, the person seems fine on the surface, yet continues to endure chronic functional problems. Some people suffer long-term effects of MTBI, known as postconcussion syndrome (PCS). Persons suffering from PCS can experience significant changes in cognition and personality.

Most traumatic brain injuries result in widespread damage to the brain because the brain ricochets inside the skull during the impact of an accident. Diffuse axonal injury (Figure 1) occurs when the nerve cells are torn from one another. Localized damage also occurs when the brain bounces against the skull. The brain stem, frontal lobe, and temporal lobes are particularly vulnerable to this because of their location near bony protrusions.

The brain stem is located at the base of the brain. Aside from regulating basic arousal and regulatory functions, the brain stem is involved in attention and short-term memory. Trauma to this area can lead to disorientation, frustration, and anger. The limbic system, higher up in the brain than the brain stem, helps regulate emotions. Connected to the limbic system are the temporal lobes which are involved in many cognitive skills such as memory and language. Damage to the temporal lobes, or seizures in this area, have been associated with a number of behavioral disorders. The frontal lobe is almost always injured due to its large size and its location near the front of the cranium. The frontal lobe is involved in many cognitive functions and is considered our emotional and personality control center. Damage to this area can result in decreased judgement and increased impulsivity.