Quote:
Originally Posted by Mark in Idaho
t97tab,
After a few days to even a few weeks of letting/waiting for these cells to heal, (six weeks is not unheard of) the damaged cells release a signal in tRNA (transmitting RNA) that shuts down the injured cells. This continues to happen until a clean margin is formed (clean margin means cells that have no damage). The healthy cells know how to ignore the shut down signal.
This is when the symptoms start showing up.
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Hi Mark! You provid great info to people in need, but you really have to explain this to me. Do you mean that damaged cells release tRNA (transport RNA, not "transmitting RNA") and that this makes injured cells nearby "deactivated"? Im just a medical student right now but I dont get what you mean.
Is it possible what you mean is that when a cell gets injured, it or its leaked content causes an inflammatory process (which can be activated by almost anything that shouldnt be in the extra cellular environment as tRNA, ATP and so forth) which causes the neuroglia to phagocytize and remove damaged neurons that still are functional and that this is the cause for the delayed onset of symtoms?