Epo OR Heparin 1 time a mounth + Pure vitamin E in a daily basis (Tocopherol ) = Cure of all traumatic TOS in 1-2 years by eradicating fibrosis. Yeah all : Neurogenic, arterial, venous.


Establised Radiation-Induced Fibrosis is cured by Pentoxifylline and Tocopherol (vitamine E) in 3 mounth- 2 years.

Proof :
http://jco.ascopubs.org/content/21/13/2545.full by dr delanian.
It is now largely accepted by doctors.

The very interesting thing is, fibrosis of TOS is the same as RIF.
Proof :

RIF :
http://jco.ascopubs.org/content/21/13/2545.full
http://books.google.com/books?id=yg6...herapy&f=false

TOS'S fibrosis :
http://www.doctorellis.com/Lecture.html
http://ukpmc.ac.uk/abstract/MED/2301...76C2F5DBA8BA1F 5.jvm1

If you read informations closely you will see it's exactly the same thing.
Dr delanan is a famous french doctor, here some study from her :

http://www.ncbi.nlm.nih.gov/sites/en...rm=%28delanian[All%20Fields]%20AND%20%28%22fibrosis%22[MeSH%20Terms]%20OR%20fibrosis[Text%20Word]%29%29

Post-op fibrosis seem to be te same thing too.
http://www.ncbi.nlm.nih.gov/pubmed/18996041

OK Now i suggest :
(A) 800 mg/d of PTX and 1,000 U/d of Vit E

BUT maybe Heparin / Epo can be more powerfull than PTX if you look at their action.

Discuss

http://www.doctorellis.com/Lecture.html


Fibrotic tissue is usually thought of as pharmacologically inert, causing problems of constriction and pain directly by mechanical strangulation of the affected nerves. The epi- or perineural fibrotic scar that the vascular surgeon was removing, however, turned out to be pharmacologically very active with multiple pro-inflammatory neuropeptides secreted by a rich matrix of largely immature neurites traversing the fibrosis. This was determined by immunoassay at UCSF and histology at UCLA. Most of the time there was little evidence of a direct mechanical effect. The closest analogous description that I could find in the literature was that of painful hypertrophic scarring, which showed a similar inflammatory neuropeptide distribution and an abundance of growth factors that were also pro-inflammatory (NGF, FGF…), at least in part.

In fact, this comparison to hypertrophic scarring did not seem far fetched when observing the extensive fibrosis often present in surgical re-dos. This, again emphasized the importance of neuronal secretions and elaborations of inflammatory substances and growth factors.
Our observations, both clinically and histologically, led to our considering NTOS as a disorder of neurogenic inflammation [...]

http://ukpmc.ac.uk/abstract/MED/2301...DBA8BA1F5.jvm1

The results revealed a consistent abnormal histologic pattern in patients with traumatic TOS: type II fibers were atrophied; there was an increase in the average number of type I fibers (78% versus 53% in muscles from control patients); and there was a significant increase (mean: 36%) in connective tissue (muscles from control patients averaged less than 15%)

Extensive work has underscored the importance of cytokine driven inflammatory processes that are Cox 1 and 2 independent as being responsible for sensitizing nerves, initiating and perpetuating much of neuropathic pain.

It is these cytokines, of which TNF alpha is probably the most notorious, that sensitize the tissues adjacent to the small initial lesions, subsequently spreading through a variety of mechanisms perpetuated by recurrent injury and inflammation.

I'm sure by 95%, i have emailed doctor. I keep you informed.