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Old 03-27-2007, 05:48 PM
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reverett123 reverett123 is offline
In Remembrance
 
Join Date: Aug 2006
Posts: 3,772
15 yr Member
reverett123 reverett123 is offline
In Remembrance
reverett123's Avatar
 
Join Date: Aug 2006
Posts: 3,772
15 yr Member
Default Clues

If PD were just a shortage of dopamine inhibiting movement, this could not happen. That it does would seem to indicate a control problem of an unusual nature. It also means that far too much money goes into levodopa research and too little into other areas.

Quote:
Originally Posted by ZucchiniFlower View Post

I expanded the use to walking and my gait is better. Instead of focusing on walking foward, I think of my walking as walking side to side, sideways. Sometimes I sway a bit. But just imagining moving sideways helps my gait a great deal.

I don't have resting tremor but my gait disorder and bradykinesia are helped by 'moving sideways' instead of foward, even thought the the motion is a foward motion. When my brain thinks sideways, I just move better, and I don't know why, but it works.
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Born in 1953, 1st symptoms and misdiagnosed as essential tremor in 1992. Dx with PD in 2000.
Currently (2011) taking 200/50 Sinemet CR 8 times a day + 10/100 Sinemet 3 times a day. Functional 90% of waking day but fragile. Failure at exercise but still trying. Constantly experimenting. Beta blocker and ACE inhibitor at present. Currently (01/2013) taking ldopa/carbadopa 200/50 CR six times a day + 10/100 form 3 times daily. Functional 90% of day. Update 04/2013: L/C 200/50 8x; Beta Blocker; ACE Inhib; Ginger; Turmeric; Creatine; Magnesium; Potassium. Doing well.
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