Interesting link; and the comments are as well.

With respect to diet, the low fat-fresh foods (raw) diet recommended in the link takes us back to Swank's diet recommendations; or a derivative there of, and that in turn would seem to take us even further back to interfering with a condition rather than with a cause. Either way, many have experienced profound benefits from the Swank type diet, while others not so much.

This sort of variance in response is what makes the auto-immune inflammatory diseases such a head scratcher for everyone concerned. While interfering with or manipulating conditions has its merits for symptom control, it does seem that the 'cure' would more likely be found in the cause (the seed itself).
Similarly, for a more reliable and universal response to potential treatment, it seems to be more reasonable to interfere with the cause; and that seems to indicate something along the lines of gene therapy holding the 'secret cure'.

Unfortunately, gene therapy is in its infancy and only recently has it been found that not only the genes themselves carry the blue print for encoded tendencies, but so does the protein coat that envelopes each and every strand of DNA. When the human genome project (DNA mapping) was being done, that vital bit of protein material was centrifuged out of the test samples and discarded. This would be like evaluating an architect's blue print but eliminating the contractor/builder who would ultimately interpret it and utilizing their own individualized 'tastes', perform the construction.
Apparently, at least according to current research in genetics, it is that protein coat that is so individual; and thus contains at least some of the information that governs how each individual person expresses each gene. The ever changing protein components within the body also seem to allow for an equal variance of expression during cell division within the same individual over time and changing conditions.

For example a person might have a gene that when activated promotes excess inflammation, but it is apparently the protein coat that determines what tissue that inflammation may primarily affect. Perhaps at one point in their life the inflammation settles in the neurological system, and perhaps later, changing conditions (diet would be an example), exert an influence on the protein coat that envelopes their DNA, and the inflammation then has a tendency to settle in their digestive tract.

This may very well be the reason that although, and thus far, no consistent genetic link has been found between family members who share a specific auto-immune disease in common. Due to having been exposed to similar conditions, they may in fact share in common the information that ultimately is expressed through the protein coat of DNA.
This gene + common denominator theory is supported by the observation that at least for MS, there seems to be a connection between race (genetic predisposition)+ climate (condition).

Confused? As I said earlier, this has been my struggle too. Just trying to get a handle on the research that has been done thus far in this field is enough to test the patience of a tortoise. Advancing the research has me taking a deep and humble bow to those who have dedicated themselves to the task.

In the end, it may very well turn out that after all the research and hypotheses are critically evaluated, and potential treatment is sent into motion that it is found to be at least partially off base.
So it has been in the case of cancer research, where after exploring genetics as a cause, some researchers have begun to give equal weight to the virus theory (cause), and many have also begun to couple that with 'contributing factors' (conditions) theory as well.

If nothing else, perhaps the auto-immune diseases like MS have served to foster greater compassion, understanding and patience within those who are involved with it.
I believe that it is so.

With love, Erika