[KimS]

Some of you may remember I was compiling information on high eosinophil levels before the obt blackout. For those who are new, the bottom line is that both dh and ds have raised e's and have had for years. No doctor has addressed this issue... or seems to be able to... and because both still have issues after going gf and multi-things free, I am looking in the e. direction for some hint of what to do next.

Also, for new people, do note that going gf and multi-things free has improved their health and quality of life by about 95%.

Okay.... summary to date is pretty quick...

I found that high e's seem, most commonly to be related to aspergillus. There is an enormous range of aspergillus and it is often found in packaged foods (another reason to stick to meat, fruit and veggies), dirt, dust, etc.

So, perhaps the next question is how to isolate which form is the biggest offender for each individual? Hmmm

Next question? How to eliminate it without making yourself crazy.... double Hmmmm...

My biggest frustration is thinking that I'm spending all my spare time researching this stuff and it might not even be our problem.

Hopefully sharing this stuff will help somebody... if not us.

Here's a link to the pillow thread... don't freak out... it's just some stuff to ponder...

Come to think of it, you should probably read this link last.
http://forums.braintalk2.org/showthread.php?t=3784

http://www.blackwell-synergy.com/doi....x?cookieSet=1

For example, such eosinophil-deficient mice were protected from airway remodelling but not allergen-induced airway dysfunction in an animal model of asthma (13). The eosinophil is involved in many inflammatory processes occurring in a variety of organs, especially the airways and the lung, where its role may be either beneficial or detrimental. For example, an eosinophil-mediated inflammatory response is usually considered beneficial during parasitic infestation, whereas eosinophilic infiltration of the bronchial wall participates to the pathophysiology of asthma. Although major advances have been made in the understanding of eosinophil biology, it remains to be determined how the different stimuli potentially induce the differential release of the variety of granule proteins and/or cytokines contained and/or synthetized by this cell type. Of note, research on eosinophil is hampered by the inability of murine eosinophils to degranulate either in vivo or in vitro, in contrast with human eosinophils, and observations issued from animal studies may not always be applicable to human disease.