[Conductor71]

Ties right into our look at other neurotransmitters in getting beyond the dopamine...

Unusual on two points:

First time an organophosphate based pesticide (widely used Malathion) has induced Parkinsonism and responded to levodopa therapy

First time an acute loss of acetylcholinesterase results in secondary Parkinsonism that is levodopa responsive. This means he had over expression of acetylcholine; why would taking levodopa help in a person who did not have low dopamine levels?

This raises questions on how perhaps a serious imbalance of one of the other catelochamines may be a trigger for Parkinson's. It is well documented that SSRI's trigger PD symptoms as well. So tipping the scales with merely one other neurotransmitter can induce Parkinsonism? In many cases the PD symptoms improve on their own but not in all.

BTW, clinically he was at stage V. How can this happen without a loss of dopamine?

Seems like we could learn from those who recover. How are our brains different or same? Can they look at something like this an animal model?

Laura

[pegleg]

Interesting, Laura. Kind of all beginning to fit together. Parkinson''s appears to be getting close to being an immune deficiency. Read this:
“Our data show that when a certain master protein that stimulates the immune system and antiviral response is expressed at high levels, it causes neuronal loss primarily in the nigrostriatal tract, thereby creating vulnerability to Parkinson’s and similar movement disorders.”
http://psychcentral.com/news/2011/05...oms/26241.html

And I am of the opinion that all of those "brain chemicals" get out of whack when our bodies are stressed - whether it be due to a virus or trauma, or whatever.. Sooo. . . why can't we start checking out such theories on a big scale? I think Fox Fdn. may be heading that way.

Just HURRY!
Peg