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04-05-2008, 03:31 AM | #1 | |||
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In Remembrance
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1: Int J Neurosci. 1994 May;76(1-2):61-9.
Neuropharmacological evaluation of movement disorders that are adverse reactions to specific foods. Gerrard JW, Richardson JS, Donat J. College of Medicine, University of Saskatchewan, Saskatoon, Canada. Three cases are reported of patients who had episodic movement disorders triggered by foods or components of the diet. In the first patient, the movement consisted of shaking the head from side to side that was triggered by milk and a number of other foods. In the second patient, the movement consisted of a repeated shrugging of the shoulders that was triggered by egg and coffee. In the third, the movement consisted of rhythmic contractions of the arms and legs that were triggered by aspartame. The first patient agreed to participate in a study in which she drank 250 ml of skim milk, an amount sufficient to trigger head shaking, after pretreatment with drugs known to alter neurotransmission across beta-adrenergic, dopaminergic, GABAergic or purinergic synapses. At the doses used, propranolol and diazepam had no effect on the milk evoked movement disorder. Levodopa (plus carbidopa) blocked the reaction to milk. Haloperidol, salbutamol and theophylline by themselves triggered a reaction similar to that evoked by milk. These observations suggest that, in susceptible individuals, foods can trigger movement disorders through an action on dopamine and other neurotransmitter pathways in the brain. A videotape of the reactions of the first two patients is available. PMID: 7960470 [PubMed - indexed for MEDLINE]
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Born in 1953, 1st symptoms and misdiagnosed as essential tremor in 1992. Dx with PD in 2000. Currently (2011) taking 200/50 Sinemet CR 8 times a day + 10/100 Sinemet 3 times a day. Functional 90% of waking day but fragile. Failure at exercise but still trying. Constantly experimenting. Beta blocker and ACE inhibitor at present. Currently (01/2013) taking ldopa/carbadopa 200/50 CR six times a day + 10/100 form 3 times daily. Functional 90% of day. Update 04/2013: L/C 200/50 8x; Beta Blocker; ACE Inhib; Ginger; Turmeric; Creatine; Magnesium; Potassium. Doing well. |
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"Thanks for this!" says: | lou_lou (04-05-2008) |
04-05-2008, 01:55 PM | #2 | ||
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And we have aspartame in Parcopa because......?????
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"Thanks for this!" says: | lou_lou (04-06-2008) |
04-06-2008, 04:05 PM | #3 | |||
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but interesting nevertheless. Can anyone think of something that cannot possibly cause movement disorders??? Every time we turn around we are faced with some new fragment of information that confirms what we all know. We have PD because we ____________(fill in the blank with whatever you want) Somebody can probably come up with a link that will support your thesis.
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I would never die for my beliefs because I might be wrong. Bertrand Russell |
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"Thanks for this!" says: | lou_lou (04-06-2008) |
04-06-2008, 04:32 PM | #4 | |||
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In Remembrance
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actually rev,
if we can not digest the food -we will have a reaction to it.. ie: How does low blood sugar occur? The intake of refined carbohydrates causes a cascade of events. Firstly, refined carbohydrates and sugars are digested quickly because of a combination of lack of nutrients and little or no fiber content. The surge of glucose into the blood stream causes the pancreas to secrete more insulin. Insulin is the hormone that carries glucose to the body’s cells so that energy can be made. It also helps store extra glucose in the liver and muscles called glycogen for later use. This overproduction of insulin lowers the blood sugar to a lower level than before food was eaten. In response the adrenal glands secrete adrenaline to bring the sugar back up by pulling stored glucose from the muscles and liver. Unfortunately, the Standard American Diet (SAD), a fitting acronym, is loaded with lots of highly processed foods that are high in sugars and fats. For many people this continual cycle of stress on their blood sugar balancing mechanisms puts them at a greater risk of developing diabetes. In essence the glandular system is eventually run down and exhausted. How is hypoglycemia treated? The best way to break the cycle of hypoglycemia and achieve stable blood sugar levels is through a combination of specific dietary changes and nutritional supplementation. A wholesome diet is the most important step. This involves eating whole, unprocessed foods on a daily basis at properly spaced intervals. Many people have too long of a gap between their meals. For example, breakfast at 6 a.m., lunch between 12 to 3 p.m. and dinner between 6 to 9 p.m. These potential long gaps can really create problems. In addition to a wholesome diet it is helpful to have healthy snacks in between your main meals. Once a person has been following a wholesome diet for a significant period of time the body re-establishes balance and sustained energy is achieved. http://www.thehealingpartnership.org...al_therapy.htm What is a wholesome diet? As far as guidelines the Glycemic Index (GI) has proven to be a valuable tool for determining how quickly particular foods effect the blood sugar. Many patients have utilized the glycemic index with great success. It is versatile and it’s beneficial for both diabetics and people with hypoglycemia. The GI of a particular food is based on factors like protein, fat and fiber content as well as degree of food processing. For example, a whole apple has a much lower GI compared to applesauce, apple juice or apple drink. Vegetables are free foods because their glycemic index is so low and they contain large amounts of fiber. Root vegetables like potatoes, carrots, beets, yams and other starch-based foods like corn are the exception and should be greatly reduced or eliminated in the diet. Fruits are generally low glycemic except for watermelon, cantaloupe, pineapple and dried fruits, which are not. Whole grains are on the lower side compared to white or wheat bread, crackers, pretzels, pastries and cookies which are all high on GI scale. Protein based foods like chicken, eggs, fish, meat are not rated on the glycemic index since the body has to work much harder to convert protein into glucose. High fat foods like ice cream and sausages, while low on the glycemic index, are nutritionally not healthy food choices. Also a diet high in saturated fats and hydrogenated oils has a more detrimental effect on blood sugar balance. For further information on the glycemic index a special handout from the office can be helpful to you. Foods allowed Foods to avoid Meat, chicken and fish Table sugar, honey Dairy products Pie, cake, pastries, cookies Low glycemic fruits and vegetables Candy, chocolate, ice cream Nuts and seeds Coffee Carob Soda, cola Whole grain bread, pasta, cereal Refined grains and cereals Slow cooking oatmeal White bread, most wheat breads Legumes Products made with white flour Unsweetened herbal teas Dried fruit Seltzer water Fruit juice Other important factors: Avoid caffeine in coffee, cola and chocolate. Caffeine induces significant stress on the adrenals, liver and digestive tract. Avoid alcohol in all forms. Alcohol is quickly absorbed and very toxic to the liver and intestinal tract. Alcohol leads to insulin resistance and large population studies have found that alcohol intake is strongly correlated with diabetes. Do not smoke, if you do you must quit. Persons with diabetes and hypoglycemia are at more risk to the negative health effects of smoking. Smoking increases the risks for diabetic complications, heart disease and stroke. Exercise A well-designed exercise program is vital for blood sugar control. Exercise makes the muscles more responsive to glucose as well as improving the overall function of your metabolism. A combination of cardiovascular exercise (walking, biking, treadmill, step aerobics) and weight training (free weights, machines, push-ups, pull-ups) will provide the best results. Join a fitness center or local health club and hire a trainer for a few sessions to get you started. We also have several books available in the Vitamin Room that have diagrams of training programs. The most important thing is to have fun with whatever plan you choose. Start out slow and allow your body to adjust. Enthusiasm is critical to your success - just don’t over do it. Follow your supplement regime faithfully Dr. Bonnet has designed a supplement regime based on your specific needs. Vitamin, minerals, amino acids, fatty acids and homeopathic remedies are critical to helping your body heal itself. Insulin resistance, a condition where the cells of the body are not able to efficiently respond to the hormone insulin, is also a well-recognized problem in relation to hypoglycemia and diabetes. This condition has been termed Syndrome X in the popular literature. Fortunately, insulin resistance responds well to dietary changes and nutritional supplements such as chromium, alpha lipoic acid, vanadium and B vitamins. A customized dietary program can also be designed through the guidance of a naturopath like myself. Wellness education and therapeutic diets really make a difference. Call the office for details and appointment times. ====
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with much love, lou_lou . . by . , on Flickr pd documentary - part 2 and 3 . . Resolve to be tender with the young, compassionate with the aged, sympathetic with the striving, and tolerant with the weak and the wrong. Sometime in your life you will have been all of these. |
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04-06-2008, 04:42 PM | #5 | |||
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In Remembrance
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they are either idiots -or the are stupid? hahahaha...
they realize it - we know -and the pharmacists know - and they should let the folks know, Definition Phenylketonuria (PKU) is a rare, inherited, metabolic disorder that can result in mental retardation and other neurological problems. People with this disease have difficulty breaking down and using (metabolizing) the amino acid phenylalanine. PKU is sometimes called Folling's disease in honor of Dr. Asbjorn Folling who first described it in 1934. Description Phenylalanine is an essential amino acid. These substances are called "essential" because the body must get them from food to build the proteins that make up its tissues and keep them working. Therefore, phenylalanine is required for normal development. Phenylalanine is a common amino acid and is found in all natural foods. However, natural foods contain more phenylalanine than required for normal development. This level is too high for patients with PKU, making a special low-phenylala-nine diet a requirement. The incidence of PKU is approximately one in every 15,000 births (1/15,000). There are areas in the world where the incidence is much higher, particularly Ireland and western Scotland. In Ireland the incidence of PKU is 1/4,500 births. This is the highest incidence in the world and supports a theory that the genetic defect is very old and of Celtic origin. Countries with very little immigration from Ireland or western Scotland tend to have low rates of PKU. In Finland, the incidence is less than 1/100,000 births. Caucasians in the United States have a PKU incidence of 1/8,000, whereas Blacks have an incidence of 1/50,000. Related diseases: Maternal phenylketonuria is a condition in which a high level of phenylalanine in a mother's blood causes mental retardation in her child when in the womb. A woman who has PKU and is not using a special low-phenylalanine diet will have high levels of phenylalanine in her blood. Her high phenylalanine levels will cross the placenta and affect the development of her child. The majority of children born from these pregnancies are mentally retarded and have physical problems, including small head size (microcephaly) and congenital heart disease. Most of these children do not have PKU. There is no treatment for maternal phenylketonuria. Control of maternal phenylalanine levels is thought to limit the effects of maternal phenylketonuria. Hyperphenylalaninemia is a condition in which patients have high levels of phenylalanine in their blood, but not as high as seen in patients with classical PKU. There are two forms of hyperphenylalaninemia: mild and severe. In the mild form of the disease, patients have phenylalanine blood levels of less than 10 mg/dl, even when eating a normal diet (0.6–1.5 mg/dl is considered the normal range). There are few effects from the mild form of the disease. In the severe form of the hyperphenylalaninemia, patients have higher levels of phenylalanine in their blood. The severe form is distinguished from classical PKU by testing for the presence of phenylalanine hydroxylase (an enzyme that breaks down phenylalanine) in the liver. Classic PKU patients lack this enzyme in their liver, while patients with severe hyperphenylalaninemia have some enzyme activity, but at greatly reduced levels compared with normal persons. Patients with severe hyperphenylalaninemia are treated with the same diet as classical PKU patients. Tyrosinemia is characterized by high levels of two amino acids in the blood, phenylalanine and tyrosine. Patients with this disease have many of the same symptoms as seen in classical PKU, including mental retardation. Treatment consists of a special diet similar to the diet for PKU. The main difference between the two diets is that patients with tyrosinemia must eat a diet that is low in both phenylalanine and tyrosine. Tetrahydrobiopterin deficiency disease is another metabolic disorder. Patients with this disease also have high levels of phenylalanine in their blood. Although phenylalanine levels can be controlled by diet, these patients still have mental retardation because they do not make enough of the neurotransmitters dopamine and serotonin, which are essential for proper neurologic function. — John T. Lohr, PhD ps - aspartame is really not an amino acid it is equal and who knows I believe it was for embalming the dead.... bigpharma is soooooooooo nice to us - arent they?
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with much love, lou_lou . . by . , on Flickr pd documentary - part 2 and 3 . . Resolve to be tender with the young, compassionate with the aged, sympathetic with the striving, and tolerant with the weak and the wrong. Sometime in your life you will have been all of these. |
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04-06-2008, 05:38 PM | #6 | |||
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In Remembrance
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A little meat, a bunch of green stuff, the occasional root. And on special occassions, filet au mammoth woolie'
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Born in 1953, 1st symptoms and misdiagnosed as essential tremor in 1992. Dx with PD in 2000. Currently (2011) taking 200/50 Sinemet CR 8 times a day + 10/100 Sinemet 3 times a day. Functional 90% of waking day but fragile. Failure at exercise but still trying. Constantly experimenting. Beta blocker and ACE inhibitor at present. Currently (01/2013) taking ldopa/carbadopa 200/50 CR six times a day + 10/100 form 3 times daily. Functional 90% of day. Update 04/2013: L/C 200/50 8x; Beta Blocker; ACE Inhib; Ginger; Turmeric; Creatine; Magnesium; Potassium. Doing well. |
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