Quote:
Originally Posted by kreink
(Post 485213)
My PN is the result of high blood sugars from Type 2 Diabetes, the PN began before I knew I was diabetic, it wasnt until I through my back out and ended up in the ER that the doc asked if I knew I was diabetic...
I am sure stress was a contributing factor to my Diabetes if for no other reason than when stressed I would turn to food and be less dilligent about excercise.
I can say that stress increases my PN pain alot. Over the summer I developed Gastroparesis and could not stop throwing up, when it was acute and I had to be rushed to the ER, it was like I could feel the burning numbness spread all the way up to mid thigh and could not stand on my feet.
This of course made me more stressed and made me throw up more and made the PN hurt more. Lol a viscous circle. Kind of a chicken/egg in that instance for me at least.
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Kevin, do you know that there is a food borne illness caused by Camylobacter that causes rapidly progressing PN? Severe GI symptoms followed by a rapidly progressing motor PN.
It is treated with IVIG.
Quote:
Turk J Pediatr. 2008 Sep-Oct;50(5):443-8.Links
Association of Campylobacter jejuni infection and GuillainBarré syndrome: a cohort study in the northwest of Iran.
Barzegar M, Alizadeh A, Toopchizadeh V, Dastgiri S, Majidi J.
Departments of Pediatrics, Tabriz University Medical Sciences School of Medicine, Tabriz, Iran.
Recent studies have suggested that Campylobacter jejuni is a common pathogen causing Guillain-Barré syndrome (GBS). This study aimed to determine the frequency and clinical and electrophysiological features of C. jejuni infection in children with GBS. We carried out a prospective study on a cohort of 48 children with GBS admitted to Tabriz Children's Hospital in the northwest of Iran from January 2003 to March 2005. Serologic investigations were used to diagnose preceding C. jejuni infection. Evidence of a recent C. jejuni infection was found in 23 (47.9%) of the patients. C. jejuni-associated GBS patients were younger than others (p = 0.010), and they had a rapid progression to reach peak disability (p = 0.018). Neither the peak disability nor the residual one-year disability was different between the C. jejuni- positive and C. jejuni-negative patients. The patients with preceding C. jejuni infection were more likely to have axonal neuropathy (p = 0.021).
PMID: 19102048 [PubMed - indexed for MEDLINE]
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from http://www.ncbi.nlm.nih.gov/pubmed/1...ubmed_RVDocSum
More here:
http://www.cdc.gov/nczved/dfbmd/dise...bacter_gi.html |
