Anyone seen this?
 

Joan IM'd this to me today,

http://news.yahoo.com/s/nm/20080323/...arkinsons_dc_1

Cloned cells treat Parkinson's in mice
By Maggie Fox, Health and Science EditorSun Mar 23, 2:06 PM ET

Researchers who used cloned embryonic stem cells to treat Parkinson's disease in mice said on Sunday they worked better than other cells.

...They put these into the brains of the injured mice. These mice got better, Tabar said.

Please check out the link for the full article.

Cloned cells treat Parkinson's in mice
 

thank you rd42 for the article and nice formatting.
My answer is in an arabic verse which says :
I live because I have hope
How hard life woud be without hope !

Hope is all that keeps me going some times.

I missed this
 

and reported same on another thread. it's all good stuff. keep it commin....

Thanks, and did you see this?
 

J Neurosci. 2007 Nov 21;27(47):12808-16. Links
Methionine sulfoxide reductase A and a dietary supplement S-methyl-L-cysteine prevent Parkinson's-like symptoms.

Wassef R, Haenold R, Hansel A, Brot N, Heinemann SH, Hoshi T.
Department of Physiology, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA.
Parkinson's disease (PD), a common neurodegenerative disease, is caused by loss of dopaminergic neurons in the substantia nigra. Although the underlying cause of the neuronal loss is unknown, oxidative stress is thought to play a major role in the pathogenesis of PD. The amino acid methionine is readily oxidized to methionine sulfoxide, and its reduction is catalyzed by a family of enzymes called methionine sulfoxide reductases (MSRs). The reversible oxidation-reduction cycle of methionine involving MSRs has been postulated to act as a catalytic antioxidant system protecting cells from oxidative damage. Here, we show that one member of the MSR family, MSRA, inhibits development of the locomotor and circadian rhythm defects caused by ectopic expression of human alpha-synuclein in the Drosophila nervous system. Furthermore, we demonstrate that one way to enhance the MSRA antioxidant system is dietary supplementation with S-methyl-L-cysteine (SMLC), found abundantly in garlic, cabbage, and turnips. SMLC, a substrate in the catalytic antioxidant system mediated by MSRA, prevents the alpha-synuclein-induced abnormalities. Therefore, interventions focusing on the enzymatic reduction of oxidized methionine catalyzed by MSRA represent a new prevention and therapeutic approach for PD and potentially for other neurodegenerative diseases involving oxidative stress.
PMID: 18032652 [PubMed - indexed for MEDLINE]

Waiter, please substitute coleslaw for the fries....
Jaye