Reflex Sympathetic Dystrophy (RSD and CRPS) Reflex Sympathetic Dystrophy (Complex Regional Pain Syndromes Type I) and Causalgia (Complex Regional Pain Syndromes Type II)(RSD and CRPS)

 
 
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Old 03-11-2012, 11:56 AM #11
Orlin1 Orlin1 is offline
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Join Date: Mar 2012
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10 yr Member
Orlin1 Orlin1 is offline
Junior Member
 
Join Date: Mar 2012
Posts: 6
10 yr Member
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First of all I am not a doctor nor have I never formally studied this , i only interested in neurologi and similar topics and i have some experince with few types of stimulation like tdcs,ces,gvs,cvs,avs.

issue around the CRPS is new for me and will me take a while before I read articles, but i may have some ideas how to help.

did you ever tried L-DLPFC stimulanin and after this C3 (or sensory) protocol ?
According to my current knowledge, it would be much more efficient.
I try to find better protocol which will combine both. This take a while i have must read lots of articles and counting electrodes size, current densities, find map of current popagaition truerh a cortex...

Before testing please consult with the doctor.

Some theory from one article :
Stimulation of the primary motor cortex (M1) and dorsolateral prefrontal cortex (DLFPC) may both reduce the perception of pain, but recent studies suggest that they likely do so by different mechanisms. Pain and perception thresholds to electrical stimulation were assessed in 20 healthy volunteers before and during anodal TDCS. Four conditions of stimulation were compared: M1, DLPFC, occipital cortex, and sham. Anodal tDCS of M1 increased both perception and pain thresholds, while stimulation of DLPFC increased pain thresholds only. The results suggested that 1) anodal stimulation of M1 but not DLPFC could induce analgesia by modulating sensory discrimination and 2) stimulation of DLPFC could modulate the perception of pain via a mechanism independent of sensory perception (21). An adjunctive study with 22 healthy volunteers showed that anodal tDCS of the DLPFC (but not M1, occipital, or sham) could decrease the perception of unpleasantness and reduces emotional discomfort/pain while subjects viewed emotionally aversive images demonstrating human pain.

some my experience i expedimentng mainly on L-DLPFC.
Stimulation of those parts of the brain lowering the levels of stress hormones in the body and activation of DLPFC alows to control emotional response and
many other suffs in the brain.From my experinece if i get the stres hormones to the zero the headache and other pain dimish (the cause of pain is still be there bud signal level to brain is lower) my thetory is taht becouse stress block endorphines withs block pain signals.If you unblock the endorphines they are block main part of pain going to the brain.(its oversmilified is very hard for me write in english).There is many posibilities how you can change how many signels go to the cortex -change thalamus settnigs or change how cortex working with signals.
The stress part of the pain can by reduced by tdcs DFLPFC stimulation or by mental technigues effect is very simmilar.

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for pain is very good suplement DLPA - dextrolevo phenylalanine
its safe without a side effects, it rise up the levels of endorphnies and some neurotransmiters in the brain

Last edited by Chemar; 03-11-2012 at 12:46 PM.
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