i started listening to an audio book

http://www.amazon.com/The-Other-Brai...he+other+brain

anyways in the far future maybe studies on glial cells could help us..or at least it sounds like they could help RSD sufferers, which i feel like i have a milder version at times.

this is why drugs targeting just the neurons do not work, supposedly


http://www.rsds.org/pdfsall/Fields_S...icAmerican.pdf


According to About.com "Glial cells are non-neural cells that perform "housekeeper" functions such as clearing out debris and excess materials. Glial cells support neurons by providing support and nutrition. There are several different types of glial cells: astrocytes, oligodendrocytes, microglia, ependymal cells, radial glial, satellite cells and schwann cells. It is estimated that there are 10 to 50 times more glial cells than there are neurons in the brain."

Key Concepts
Chronic ■ ■ pain that persists
after an injury heals is
often caused by overly
excited pain-sensing neurons
that signal without
an external stimulus.

■ ■ Traditional pain drugs that
target neural cells directly
rarely quiet these abnormal
pain messages because
the neurons’ heightened
sensitivity is driven
by a different type of cell
called glia.

■ ■ Such cells monitor the
activity of neurons and
attempt to keep them
healthy and functioning
efficiently. But well-intentioned
glial reactions to
intense pain can at times
prolong that pain.

Does that explain hypersensitivity due to the spasming of the cervical sympathetic ganglion?
As my scar tissue is being worked on and my therapist is desensitizing it , my hypersensitivity is going away.

i am not sure! but it makes you think

i am hyper sensitive as well but getting better thru pt/time passing

Old stuff. Check my threads

you lost me at hypocortisolism, no offense

No problem

But i'm even more convinced about this today.

For exemple in tinnitus sufferers, they all have hypocotisolism, and the explanation may be that an early stress created it and this imper the anti-inflammatory effect of cotisol, change behaviors, and these things create autoimmune and stress diseases like tos and tinnitus (Bad behaviors + lack of antiinflammatory effect from cortisol)

To me TOS is the darwin's law : The cost of adaptation.

And often wild animals get sick from an early chronic stress, but not from diseases that kill.

It is likely that hypocortisolism is an adaptation to a stressful environment, particularly social because we are social animals, and that autoimmune/stress diseases are the result of this adaptation.

Later, additional challenges can increase behavior problems : the need to belong, intrasexual competition etc

i actually had my cortisol checked, 3x a day via saliva..and it was normal (a bit high in the morning from bad sleep/insulin resistance). which makes sense because my body temps are pretty stable. ive read people with cortisol issues have wildly fluctuating temps.

but during a flare, i have never checked.

it's not the basal level per se, it is the level of cortisol after stress.

Instead of going up it go down.