[Electra]

I just had a profound moment for me and my quest for answers. Truly boggles my mind, but I'll get wrapped around it in a day or so. I posted on BT about a month ago my thougths on CD. [I post now there, but it is SO SLOW, something is wrong.]
http://brain.hastypastry.net/forums/...ad.php?t=13667
They (the medicial community) don't really have answers and for me the disease just does not "fit" logic. Something is missing. Why is the body turning on itself in presences of gluten protien?
My FIL just sent me this link to GlycoScience.org He's a message therapist who has been in the alternative medicine side of things since long before I knew him. His library collection is fabulous living on the grid kind of stuff. Anyway...
If you have researched CD, the other related diseases and just continually seek answers, read this and just think for a moment what it implies, it isn't an answer, it's just a theory that emerges to me.

Metabolic disorders can alter glycosylation and cause adverse effects. For example, galactosemia (build up of galactose) results from deficiencies in the enzymes that convert galactose into usable metabolic forms. Serious medical consequences occur because of an inability to synthesize essential galactose-containing glycoconjugates.99 108 Decreased activity of the enzyme, galactosyltransferase, which glycosylates (galactose) immunoglobulin G, may be important in the etiology of rheumatoid arthritis. There is a lower content of galactose in glycoconjugates in both serum and synovial fluid in arthritic patients.109 110 111 Abnormal fucosylation of acute phase serum proteins has also been observed in patients with rheumatoid arthritis.19 80 Deficiency in fucosidase will lead to a disease termed fucosidosis due to accumulation of excess fucose glycoconjugates.100 101 Activities of liver fucosyltransferase and serum fucosidase are also increased in diabetic rats.80 Serum fucosidase activity is decreased in cystic fibrosis patients.19 In a rare disease termed leukocyte adhesion deficiency II, there is severely reduced fucosylation of glycoconjugates that results in a markedly compromised immune system.112 Patients with carbohydrate-deficient glycoprotein syndrome type I (CDGS) underglycosylate many serum proteins by failing to add entire N-linked oligosaccharide chains. The metabolic defect in most cases is a loss of phosphomannomutase activity. The disease can be corrected simply by supplementation with mannose.7 N-acetylglucosamine appears to be important in glycosylation reactions in the developing intestine of animals, since its incorporation into rat epithelial cell surface glycoproteins was reduced in newborns reared by mothers given low-protein diets

Does it jump out at you as it did to me? What jumped out is more questions than answers, but a theory in my head that some key sugar and sugar synethsis is missing or gone hay wire. And it MIGHT BE a key to my answers regarding Celiac Disease. Of course, I need to do more reading, more understanding, and more research b/c frankly this reads like a bio-chem text and that was just too many years ago for me to have total understanding on a first pass. I passionately loved science classes in high school and college, but choose to go a different path b/c I didn't want to spend years in research to have the PhD guy get all the credit for his grunts research. I became an accountant instead.

Anyone want to truly cure Celiac Disease with me? Alternative minded people will probably get to it before a medical doctor, they got to the diet first (CSD, then GF) that fixed the immediate problems. Now it's time for those alternative minded to come up with a theory of the reason why and then work toward a real cure.