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03-23-2008, 09:45 PM | #1 | |||
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Joan IM'd this to me today,
http://news.yahoo.com/s/nm/20080323/...arkinsons_dc_1 Cloned cells treat Parkinson's in micePlease check out the link for the full article.
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_________________________________________________ http://calipso-pd.org ...bringing a new wave of Parkinson’s support to central Illinois Last edited by rd42; 03-24-2008 at 08:15 AM. Reason: per nt guidlines re: copyright |
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"Thanks for this!" says: | Howardh (03-25-2008), imark3000 (03-24-2008), Lasgo (03-25-2008), LING (03-24-2008), ZucchiniFlower (03-24-2008) |
03-24-2008, 05:04 AM | #2 | ||
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thank you rd42 for the article and nice formatting.
My answer is in an arabic verse which says : I live because I have hope How hard life woud be without hope !
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Imad Born in 1943. Diagnosed with PD in 2006. |
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03-24-2008, 08:16 AM | #3 | |||
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Hope is all that keeps me going some times.
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_________________________________________________ http://calipso-pd.org ...bringing a new wave of Parkinson’s support to central Illinois |
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"Thanks for this!" says: | imark3000 (03-24-2008) |
03-25-2008, 03:54 PM | #4 | |||
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and reported same on another thread. it's all good stuff. keep it commin....
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The true leaders of today are those who strive for a world where it's every citizen can enjoy the benefits of scientific enlightenment and technological progress. GO HARD>>>>SCIENCE |
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03-26-2008, 12:07 PM | #5 | ||
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J Neurosci. 2007 Nov 21;27(47):12808-16. Links
Methionine sulfoxide reductase A and a dietary supplement S-methyl-L-cysteine prevent Parkinson's-like symptoms. Wassef R, Haenold R, Hansel A, Brot N, Heinemann SH, Hoshi T. Department of Physiology, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA. Parkinson's disease (PD), a common neurodegenerative disease, is caused by loss of dopaminergic neurons in the substantia nigra. Although the underlying cause of the neuronal loss is unknown, oxidative stress is thought to play a major role in the pathogenesis of PD. The amino acid methionine is readily oxidized to methionine sulfoxide, and its reduction is catalyzed by a family of enzymes called methionine sulfoxide reductases (MSRs). The reversible oxidation-reduction cycle of methionine involving MSRs has been postulated to act as a catalytic antioxidant system protecting cells from oxidative damage. Here, we show that one member of the MSR family, MSRA, inhibits development of the locomotor and circadian rhythm defects caused by ectopic expression of human alpha-synuclein in the Drosophila nervous system. Furthermore, we demonstrate that one way to enhance the MSRA antioxidant system is dietary supplementation with S-methyl-L-cysteine (SMLC), found abundantly in garlic, cabbage, and turnips. SMLC, a substrate in the catalytic antioxidant system mediated by MSRA, prevents the alpha-synuclein-induced abnormalities. Therefore, interventions focusing on the enzymatic reduction of oxidized methionine catalyzed by MSRA represent a new prevention and therapeutic approach for PD and potentially for other neurodegenerative diseases involving oxidative stress. PMID: 18032652 [PubMed - indexed for MEDLINE] Waiter, please substitute coleslaw for the fries.... Jaye |
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