This really may explain a lot about the PD personality. We have a 33-9 ratio now. This is about how we look at our outer life - whether we are organized and in control or more flexible and adaptable.

here are some links and information about these two traits

http://changingminds.org/explanation...perceiving.htm

Judging and Perceiving are preferences used in the Jungian Type Inventory. The naming is unfortunately a bit archaic as judging is more than evaluation and intuiting is not about looking at thing.

They are about how we approach life: in a structured way or an open, flexible way.

Judging
Judgers approach life in a structured way, creating plans and organizing their world to achieve their goals and desired results in a predictable way.

They get their sense of control by taking charge of their environment and making choices early.

They are self-disciplined and decisive, and seek closure in decisions. When they ask for things they are specific and expect others to do as they say. They enjoy being experts.

At work, they decide quickly and clearly and work to get the job done.

Perceivers may see them as rigid and opinionated.

Perceiving
Perceivers perceive structure as being more limiting than enabling. They prefer to keep their choices open so they can cope with many problems that the know life will put in their way.

They get their sense of control by keeping their options open and making choices only when they are necessary.

They are generally curious and like to expand their knowledge, which they will freely acknowledge as being incomplete. They are tolerant of other people's differences and will adapt to fit into whatever the situation requires.

At work, they tend to avoid or put off decisions and like most the exploration of problems and situations.

Judgers may see them as aimless drifters.


http://www.myersbriggs.org/my-mbti-p...perceiving.asp

http://www.mypersonality.info/person...ng-perceiving/

Step One-
Exposure to LPS in womb produces an anxious adult

1: Behav Brain Res. 2004 Sep 23;154(1):63-9.

Endotoxin exposure in early life alters the development of anxiety-like
behaviour in the Fischer 344 rat.

Walker FR, March J, Hodgson DM.

Laboratory of Neuroimmunology, School of Behavioural Sciences, University of
Newcastle, Newcastle 2308, NSW, Australia. rohan.walker@newcastle.edu.au

Previous research in the rat has demonstrated that neonatal exposure to
bacterial endotoxin alters the level of anxiety-like behaviour displayed in
adulthood. Currently, however, little is known about the emergence and
development of this type of behaviour. Given the ability of neonatal endotoxin
exposure to alter neural substrates involved in regulating anxiety, we tested
the hypothesis that it may also alter the developmental trajectory of
anxiety-like behaviour in the rat. Male Fischer 344 neonatal rats were treated
with endotoxin (0.05 mg/kg lipopolysaccharide from Salmonella enteriditis) or
vehicle on postnatal days 3 and 5. Age related changes in anxiety-like behaviour
were subsequently investigated using the elevated plus maze apparatus at three
developmental time points; adolescence (43 days), adulthood (80 days) and
senescence (400 days). Neonatal endotoxin exposure was found to significantly
increase circulating levels of corticosterone on postnatal days 3 and 5 at 4 h
postadministration (P < 0.05). Additionally, endotoxin exposure was found to
markedly alter anxiety-like behaviour in adulthood and senescence (P < 0.05).
Specifically, adult and senescent endotoxin treated animals displayed
significantly more anxiety-like behaviour than vehicle treated controls.
Interestingly no significant differences in anxiety-like behaviour were observed
between treatment groups during adolescence. These findings highlight the
importance of the early life microbial environment in the development of
emotional behaviour and suggests that neonatal infection may be an important
predictor of susceptibility to anxiety related disorders in adult life.

PMID: 15302111 [PubMed - indexed for MEDLINE]


Step two: Anxious adult finds relief in a sense of control


1: Biol Psychiatry. 2008 Oct 15;64(8):701-7. Epub 2008 Jun 24.

Effects of perceived control and cognitive coping on endocrine stress responses
to pharmacological activation.

Abelson JL, Khan S, Liberzon I, Erickson TM, Young EA.

Department of Psychiatry, Trauma, Stress and Anxiety Research Group, Molecular
and Behavioral Neuroscience Institute, University of Michigan, Ann Arbor,
Michigan, USA. jabelson@umich.edu

BACKGROUND: The hypothalamic-pituitary-adrenal (HPA) axis may mediate negative
health effects of stress. It is sensitive to cognitive/emotional factors like
novelty, perceived control, and coping. Psychological intervention that reduces
novelty and enhances cognitive coping and sense of control can reduce cortisol
responses to pentagastrin, a pharmacological HPA activator. This study attempted
to identify the core factors that modulate HPA axis activity in this model.
METHODS: Varying instructions were administered prior to drug exposure in a
two-visit (placebo first) pentagastrin infusion paradigm. Healthy subjects (n =
40) were randomly assigned to one of four instruction groups: 1) standard
instruction (SI); 2) full cognitive intervention (CI); 3) the CI control
component alone; or 4) the CI novelty reduction/coping components alone. Blood
samples were obtained via intravenous catheter before and after pentagastrin.
RESULTS: Subjects receiving an intervention had smaller cortisol responses than
subjects receiving standard instructions. Coping alone had as strong an impact as
the more complex intervention that combined coping and control. Control alone
also reduced cortisol but its HPA impact appeared less robust. CONCLUSIONS: Brief
psychological manipulation can significantly reduce HPA activation in challenge
paradigms. Cognitive preparation that focused on side effects, reduced potential
surprise, and enhanced cognitive coping modulated HPA axis activity as
effectively as a previously tested intervention that combined coping and control
manipulations. A sense of control alone also reduced cortisol release. The
results support development of control or coping techniques to combat negative
health effects of stress that are mediated by HPA axis activation.

PMCID: PMC2579765 [Available on 2009/10/15]
PMID: 18571624 [PubMed - indexed for MEDLINE]

Could our parents have helped mould the personality?

I was thinking about this the other day. There has to be a tendency somewhere for the condition, but in addition to this, I was wondering if our parents have helped to mould our personality into the direction we have.

My parents were always on my case about perfection. Do things at 1000%-plus or don't do it at all. You never want to make a fool of yourself. Then when I worked really hard at something. Got the standing ovation at the piano recital, etc. I was told I would never be good enough because I don't play like Horowitz or Rubenstein. So I'd go home and practice, and be told that I practice too much and I should play more! So I'd play more and be told that I don't practice enough?

The building of self-doubts, the psychotic random flips between being helpful and not, the focus on perfection, which for me left my literally paralysed my whole life because I for on never felt I was capable of doing anything at the perfect level, and two I'd wait so long I'd miss an opportunity of a lifetime that I would have really enjoyed or gotten ahead with.

The stiff upper lip old Yankee. Suck up to the pain and go on... My parents would tell me to stop whinging about an injury, toothache, earache, etc. because they didn't want to hear it. In fact my dad would tell me he was going to give me a reason to cry. This wasn't just a slap on the backside. This really was painful from what I can remember.

So rather than complain about the sore foot, earache, etc. I'd suck it up and continue on until the pain went beyond the normal 1-10 range. Mine is more like 20-30 on the normal scale, and when I complain now the agony level would put most people into the emergency center in less than a mouse-click.

Sometimes I wonder...

John

NOW we are getting to my original intent for this thread. Look back at the first post and compare these two screwed up little kids and their screwed up parents. I already know that jcitron and I are far from alone here. My question is how far does it go?

John,
Leaving out the details, but keeping in the process of upbringing, I recognise exactly what you are saying, as well as these words:

I saw an acupunturist for a year before getting my dx, and he defined my condition as being 'stuck', in all sorts of ways I could see that was what I had become, and the manifestation of it in my body was the evidence.

Not only was I so conditioned to being in that paralysed place, I was almost immune from recognising it because it was my reality. So I would walk into situations over and again that most people would not even consider......

PD has in some ways taught me what I should not be, so I am not compounding that conditioning. And in some ways things are better, even though I have PD to deal with.

Thanks for your post

Lindy

Rick,

i agree it's been bad at times and i have this inner child that never gets to play, so it leaks out in strange ways. it's a contributor but wouldn't there be just as many variations in this domain as in the motor and chemical /drug/ domains? hard to pin down? in the womb?

and now, we have neurons in our b---- lol
olsen posted something on it.

That's something we should all be glad we found out.. but most of it had been discussed here more than a year - 2? ago.

Question [pdonline research?] does that mean a treatment such as duodopa could kill off our bodies' natural capacity in anyway in the gut to operate correctly?

Well we are quite the machines...
paula

i wonder what might show up if we divided this group into tremor or rigidity dominant PD?

ENFJ - I'm rigidity dominant

paula

I did the test and was labelled:

INFJ

I havent read the other reactions to see if there is a pattern. I'm curious.

Interesting!!

Joop

me, too, bluedahlia. not surprising in light of my pleasure in viisiting the art siite - THANKS ! ibby

conversion disorder

http://emedicine.medscape.com/article/287464-overview

esp:
Psychogenic movement disorders
Conversion disorder can imitate the entire spectrum of movement disorders and include tremor, chorea, myoclonus, dystonia, tics, parkinsonism, knee buckling, and a host of other bizarre gait disturbances. A commonly used term for a type of this last phenomenon is an astasia-abasia gait pattern, in which the patient makes wild movements of the trunk and arms during a gait evaluation but does not fall or err from a stressed gait such as a tandem or toe gait.

http://www.minddisorders.com/Br-Del/...-disorder.html

esp:
Conversion disorder may also develop in adults as a long-delayed after-effect of childhood abuse.

Psychogenic movement disorders . These can mimic myoclonus, parkinsonism, dystonia, dyskinesia, and tremor. Doctors sometimes give patients with suspected psychogenic movement disorders a placebo medication to determine whether the movements are psychogenic or the result of an organic disorder.