I finally got the report back from my studies at CC. Just want to share results from both sets of tests and see what comments you all have......

1/11/2010 (@ I.U.) Impression: The above EMG / NCS reveal electrical evidence of a mixed axonal and demyelinating periperal polyneuropathy, mild in degree. There was no electrical evidence of superimposed cervical/lumbosacral polyradiculopathy, myopathy or mononeuritis multiplex.

7/26/10 (@ Cleveland Clinic) Extensive electrodiagnostic examination of the R upper and lower extremities show no abnormalities except for a mildly reduced amplitude of the H response and mild chronic motor axon loss changes isolated to the EDB. These findings are too limited for definitive diagnosis, but may be suggestive of a S1 radiculopathy that is mild in degree electrically. No active motor axon loss changes are seen. In addition, there is no evidence of a generalized sensorimotor polyneuropathy.

So now I am really confused....... See next post for skin biopsy results.

Altho the word demyelinating is mentioned, it seems to lean towards a general sensorimotor polyneuropathy....I wonder if IVIG is an option for you? I would think a case could be made for CIDP? Makes me wonder if something is post-infectious......have you had a Lyme test with Igenex? They do IgM response to Bb.

Hi there Cyclelops.....I do remember the doc at CC talking about CIDP. Basically I think he ruled it out because I had 5/5 muscle tone, no atrophy. Said I was "too strong." As far as the Lyme test, I did have the Lyme G (IgG) and the Lyme M (IgM) done but it was through ARUP Labs. Does that count? I tell you, somedays my head just swims with all of these test results in front of me. Some days it all makes sense and other days it all reads Greek.

--CIDP variants that are predominantly or exclusively sensory in nature (though the "classic" CIDP paradigm involves motor dysfunction):

http://neuromuscular.wustl.edu/antib...mdem.html#cidp

There could also be a "co-morbidity" situtaion--no one ever said that one couldn't have some radiculopathy from spinal situations and not also have a more peripheral process going on as well (and the "double crush hypothesis" theorizes that in such cases a nerve tract compromised twice will exhibit symptoms that are more than the sum of those that could be expected from the individual parts).