This is quite interesting.

Especially since my 'cholesterol' levels are not that bad (just barley over normal). Yet I have known "extensive" (word used on scan results) calcification throughout my arteries. They have no doubt that the bigger culprit is the inflammation from Sjogren's. As explained to me the inflammation makes possible for the cholesterol to attach (I would guess like you mentioned, 'patch'). This continuous process though causes the layers of endothium (I think this is the word he used) tissue to thicken and calcify. Does this make any sense??

The logic the doctor used for short term statin treatment (hoping to limit it to two months) was based upon a completely different test, not standard lipids. It was Apo B (apolipoprotein B), which is supposed be more reliable in determining the risk for plaque in the arteries. I have tried to read about this lipprotein, but really don't understand it. If you've heard of this, I'd love to hear your comments (in laymen terms).