[boytos]

Epo OR Heparin 1 time a mounth + Pure vitamin E in a daily basis (Tocopherol ) = Cure of all traumatic TOS in 1-2 years by eradicating fibrosis. Yeah all : Neurogenic, arterial, venous.


Establised Radiation-Induced Fibrosis is cured by Pentoxifylline and Tocopherol (vitamine E) in 3 mounth- 2 years.

Proof :
http://jco.ascopubs.org/content/21/13/2545.full by dr delanian.
It is now largely accepted by doctors.

The very interesting thing is, fibrosis of TOS is the same as RIF.
Proof :

RIF :
http://jco.ascopubs.org/content/21/13/2545.full
http://books.google.com/books?id=yg6...herapy&f=false

TOS'S fibrosis :
http://www.doctorellis.com/Lecture.html
http://ukpmc.ac.uk/abstract/MED/2301...76C2F5DBA8BA1F 5.jvm1

If you read informations closely you will see it's exactly the same thing.
Dr delanan is a famous french doctor, here some study from her :

http://www.ncbi.nlm.nih.gov/sites/en...rm=%28delanian[All%20Fields]%20AND%20%28%22fibrosis%22[MeSH%20Terms]%20OR%20fibrosis[Text%20Word]%29%29

Post-op fibrosis seem to be te same thing too.
http://www.ncbi.nlm.nih.gov/pubmed/18996041

OK Now i suggest :
(A) 800 mg/d of PTX and 1,000 U/d of Vit E

BUT maybe Heparin / Epo can be more powerfull than PTX if you look at their action.

Discuss

http://www.doctorellis.com/Lecture.html


Fibrotic tissue is usually thought of as pharmacologically inert, causing problems of constriction and pain directly by mechanical strangulation of the affected nerves. The epi- or perineural fibrotic scar that the vascular surgeon was removing, however, turned out to be pharmacologically very active with multiple pro-inflammatory neuropeptides secreted by a rich matrix of largely immature neurites traversing the fibrosis. This was determined by immunoassay at UCSF and histology at UCLA. Most of the time there was little evidence of a direct mechanical effect. The closest analogous description that I could find in the literature was that of painful hypertrophic scarring, which showed a similar inflammatory neuropeptide distribution and an abundance of growth factors that were also pro-inflammatory (NGF, FGF…), at least in part.

In fact, this comparison to hypertrophic scarring did not seem far fetched when observing the extensive fibrosis often present in surgical re-dos. This, again emphasized the importance of neuronal secretions and elaborations of inflammatory substances and growth factors.
Our observations, both clinically and histologically, led to our considering NTOS as a disorder of neurogenic inflammation [...]

http://ukpmc.ac.uk/abstract/MED/2301...DBA8BA1F5.jvm1

The results revealed a consistent abnormal histologic pattern in patients with traumatic TOS: type II fibers were atrophied; there was an increase in the average number of type I fibers (78% versus 53% in muscles from control patients); and there was a significant increase (mean: 36%) in connective tissue (muscles from control patients averaged less than 15%)

Extensive work has underscored the importance of cytokine driven inflammatory processes that are Cox 1 and 2 independent as being responsible for sensitizing nerves, initiating and perpetuating much of neuropathic pain.

It is these cytokines, of which TNF alpha is probably the most notorious, that sensitize the tissues adjacent to the small initial lesions, subsequently spreading through a variety of mechanisms perpetuated by recurrent injury and inflammation.

I'm sure by 95%, i have emailed doctor. I keep you informed.

[tied]

Blood thinners would logically work on venous TOS by allowing blood to go through an ever narrowing channel at the brachio plexus. If the compression got narrower I would think there would be a point of diminishing returns. But what about the compressed nerves and lymphatic system? Do blood thinners work on those? One diagnostic test which we could all perform and which doctors sometimes skip is to measure our height. Our nutritional program should not neglect conserving our bone density as we grow older.

[boytos]

Originally Posted by tied

Blood thinners would logically work on venous TOS by allowing blood to go through an ever narrowing channel at the brachio plexus. If the compression got narrower I would think there would be a point of diminishing returns. But what about the compressed nerves and lymphatic system? Do blood thinners work on those? One diagnostic test which we could all perform and which doctors sometimes skip is to measure our height. Our nutritional program should not neglect conserving our bone density as we grow older.

Heparin and Pentoxifylline blood functions are usless, they are primilary TNFA and citokyne inhibitor.

[boytos]

Radiation induced fibrosis (RIF) = TOS fibrosis
RIF is cured by Pentoxifylline (anti TNFA) and Tocopherol (ANTIOXYDANT) (vitamine E) in 3 mounth- 2 years

SO RSI TOS TOO !

All RIF (SAME AS TOS SCALENUS FIBROSIS !!! ) areas improved rapidly with regard to
local pain after withdrawal of analgesic drugs. All RIF areas
exhibited various but evident softening of the tissues
involved with a regression of fibrosis adhesion to underlying
tissues. Telangiectasias were difficult to assess, but
their number and density seemed stable. Patients with
two RIF zones displayed a proportional mean decrease in
both zones.


http://www.google.com/url?sa=t&sourc..._dG7MCH9oLYy3A

http://www.google.com/search?q=Kinet...ient=firefox-a

[boytos]

SOD seemsto be good too :

Well and long established radio-fibroses have been treated successfully with a liposomal encapsulated bovine copper superoxide dismutase. After a short treatment (three weeks intramuscular injection of 5 mg twice a week) regression of the fibrosis is stable. The average size is reduced by one third and significant softening occurs in 82% of the cases. Efficiency is independent of the time between radiotherapy (origin of the fibrosis) and treatment with liposomal SOD. Complete regression even after this limited treatment is seen in cases of chronic prefibrotic inflammatory syndromes and prophylactic action in cases where the probability of fibrosis formation is certain appears to be successful. The roles of superoxide and superoxide dismutase are discussed.

Read More: http://informahealthcare.com/doi/abs...15768609051643