We are in that group. I have often wondered about this myself....for this reason: my mother in law nursed her first son, my husband's older brother, but not his sister or him, the middle and youngest, respectively. The sister got breast cancer recently, and my husband got young onset PD. The older brother who was breastfed? He's fine.

I wonder also if the horrible formulas that are used for babies come into play. It would be one thing if your baby didn't get mother's milk, but got something very close to it. But if you read those baby formula labels, they are horrible, and filled with sugars. I can't help but think that starting innocent lives out with such awful nutrition at a time when their body NEEDS all the things in breastmilk doesn't cause untold developmental damage.

Yep, went sraight to cow's milk, too.

An interesting thread but I am pretty sure that my husband (PWP) was breast fed.
As for heart disease/stroke there are a lot of researchers who would point to all forms of sugar(anything ending in ose) as the culprit for many modern western diseases. Certainly,obesity seems to be linked to high sugar consumption. The fats to avoid as far as I am concerned seem to be those that have been developed by humans.

Sorry, but I found that last sentence quite funny. We humans seem to muck things up, don't we? As if olive oil and coconut oil were not good enough, we get in the lab to make frankenoil, hydrogenate this and that, emulsify any and everything, and who knows really just how bad that is for us.

That could be a funny bumper sticker, though, if you could word it just right.

I'm hearing a rap song ....lolg

YES to this post and, the whole cultural practices and beliefs surrounding pregnancy aand birth when my mother conceived me was ANTI feminine!

Speaking to the Parkinsons Resource Org on the phone about TMJ
adjustments the subject came up about forrcep deliveries common to PWP - my mother took DES (Diethylstiburol - a drug to prevent miscarriage) at 6 mo gestation with me - no breast milk for me - both my older brother and sister were brreastfed tho my ssister does suffer with epilepsy

I have been passionate about how i wanted to birth my kids and it was that journey that led me to learn all about health and complimentary medicine - we all are healing our ancestral heritage - guess I'm not as LOST as I thought

I would add that thhe event of chronic disease has mushroomed after massive . vaccinations became regular practice ..i know i know...just sayin........

md

QUOTE:

"Speaking to the Parkinsons Resource Org on the phone about TMJ
adjustments the subject came up about forrcep deliveries common to PWP"

Mother told me that my head was bruised with skull being mashed-in from the force the doctor used yanking me out of the womb. The more we learn about PWP it seems some things in common are strikingly the same. Wonder if this practice is still being used? I pray that it is not done as common practice anymore.

The TMJ guards the airway and governs blood flow through the carotid arteries. There is a nervous system unique to the surrrounding of the heart and the trigeminal nerve which feeds signals to tthe brain stem. Its amazing to me how resilient we are and that you could survive a bruised and mashed skull at birth is testimony to such strength and flexibility. My goodness just me!!! JUST????i dare say....you are a very brave surviivor!!!time to put on your aphrodite dress dear!!!

birthing is thankfully undergoiing a quiet revolution. All three of my kids were bron in my home midwifed through the loving hands of a very wise and capable midwife with small hands...sorry guys if you havee to uuse forceps your hands (and egos) are too big..

kind regards,
md
b

Well, I have an answer on how much saturate fat is too much when one already has chronic inflammation...there is no limit, apparently. This dietary approach is advocated by a scientist on his blog. Note toward the bottom of the list he says to restrict dietary saturated fats (that means no coconut oil). However, toward the end of his comments, he says that in two years since he wrote that post, he sees no reason to restrict the amount of saturated fats.

I am inclined to agree with Lindy on the moderation point.


Anyway, posting his anti-inflammatory diet here.

Instinctively, the inflammatory process seems to me to be a likely culprit in some of our PD symptoms. I started investigating CMO (cetyl myristoleate), a medium-chain fatty acid, for pain and indeed it seems to have helped me greatly with that.
This is a fat discovered by one scientist, Dr. Diehl, that has been studied for arthritis use.

"Clinical Observations and Usage" from
http://www.tldp.com/issue/168/168cetyl.html

In common with many other natural substances and drugs, the exact mechanism of cetyl myristoleate's physiologic activity is unclear. As a fatty acid ester, it appears to have the same characteristics as the essential fatty acids, linoleic and alpha linolenic acids, except stronger and longer lasting. These fatty acids are referred to as "essential fatty acids" because the human body cannot make them and we must ingest them in our diets. These EFA's truly are essential to normal cell structure and body function and function as components of nerve cells, cell membranes, and hormone-like substances known as prostaglandins. Many of the beneficial effects of a diet rich in plant foods is a result of the low levels of saturated fat and the relatively higher levels of EFA's. While a diet high in saturated fat has been linked to many chronic diseases, a diet low in saturated fat but high in EFA's prevents these very same diseases.The use of EFA's over an extended period of time has been shown to decrease the pain, inflammation, and limitation of motion of arthritis.

The difference between the activity of EFA's and cetyl myristoleate is that the quantity required and the period of time over which EFA's are taken are markedly longer. Cetyl myristoleate is taken in a one month course of about 13 grams, while EFA's must be taken over extended periods, sometimes many years, and intake varies widely from hundreds to thousands of grams. Cetyl myristoleate seems to have properties in common with EFA's, but it acts faster and lasts longer.

Because EFA's are necessary for normal functioning of all tissue, it is not surprising that the list of symptoms of EFA deficiency is a long one. In chronic inflammatory processes, the supply of EFA's is depleted. Cetyl myristoleate appears to have the ability to correct the imbalance created by chronic inflammation. Like EFA's, maybe cetyl myristoleate turns off the fires of chronic inflammation by serving as a mediator of prostaglandin formation and metabolism.

Venous blood from the gastrointestinal tract is carried to the liver via the portal vein. With the exception of intestinal chylomicrons that enter the lymphatics, all absorbed products pass initially through the liver, and in most instances are extracted or modified before passage into systemic circulation. Since all fatty acids enter systemic circulation through the liver, an oil like cetyl myristoleate would begin its systemic circulation from the liver also. It is speculated that cetyl myristoleate stimulates the production of immunoglobulins and series 1 and 3 prostaglandins, which could be one explanation for why cetyl myristoleate has such potent effect in auto-immune and inflammatory conditions."


Also,

MECHANISMS OF CMO ACTION AND INDICATIONS
(Written by Dr Chuck Cochran, D.C.)
The exact mechanisms of Cetyl Myristoleate action are not fully understood. Several theories have been presented, but as of today, there has been no research in this regard. Being a fatty acid ester, one mechanism being presented is that Cetyl Myristoleate somehow manipulates the production of the favorable prostaglandins, (series 1 and/or 3) and leukotrienes over the unfavorable prostaglandins of the 2nd series and pro-inflammatory leukotrienes. Prostaglandins and leukotrienes are unsaturated fatty acids that regulate many local metabolic processes including inflammation, platelet aggregation, pain, fluid balance, and nerve transmission. These effects could be accomplished by inhibition of the arachidonic acid cascade and the cyclo-oxygenase and lipoxygenase pathways.

Another mechanism being discussed is that these CMO fatty acid esters are somehow incorporated into the phospholipid cell membranes and alter cell membrane permeability and receptor sites. This could explain the possible theory of altering T-lymphocyte function during the hyper-immune response related to autoimmune diseases. Although the mechanisms are unknown, we can clinically observe Cetyl Myristoleate’s effects.
Cetyl Myristoleate seems to function in at least four different ways. One of the first observations noted when favorable results are seen is the lubricating quality of Cetyl Myristoleate. Decrease or loss of morning stiffness is commonly noted shortly after commencing CMO treatment. Next, Cetyl Myristoleate functions as an anti-inflammatory. Lessening of swollen digits is often seen after the 4th or 5th week of Cetyl Myristoleate treatment. Third, Cetyl Myristoleate functions as an immunomodulator or immune system regulator. Cetyl Myristoleate’s ability to regulate or calm down hyper-immune responses is one of the most exciting qualities and shows that Cetyl Myristoleate may be helpful in addressing the symptoms related to many autoimmune diseases. And finally, Cetyl Myristoleate functions as an analgesic or painkiller and CMO has been helpful for many sufferers of muscle tension headaches and fibromyalgia.

In any case, I am using coconut oil (about 1 T. daily) and olive oil along with supplements of evening primrose oil, a t. of cod liver oil and Co-Q10.
Heart problems do NOT run in my family but my HDL/LDL levels are always excellent.